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This drug is unique (as far as I know) from this 5a-reductase, an enzyme that converts testosterone into extremely powerful DHT, actually converting nandrolone into a less powerful compound. Thus AAS is almost ineffective on the skin, skin and prostate, and these tissues achieve lower androgen levels more efficiently than the rest of the body. Therefore, with equal activity as an alternative to androgen receptors (AR) for muscle tissue, Deca provides less activity on the skin, dermis and prostate. Therefore, the best choice for those who are most concerned about these things. Its effect on the androgen receptor on muscle tissue is greater than that of testosterone: it binds better. However, it provides only half the results of building muscle per milligram. I think this is the result of inactive or complete inactivity in non-AR pathways of muscle development. It also appears to be inactive or completely inactive in nerve cells, certainly in the nerve cells responsible for the function of the p3nis. Use of Deca as the sole AAS often leads to complete s3xual dysfunction. These problems can be solved by combining a drug that provides non-existent functions: For example. Testosterone also shows an antidote for nandrolone progestin. This fact is clearly important in bodybuilding, since only limited use of DICA actually lowers the levels of natural testosterone resulting in lower estrogen levels and thus allows less aromatic enzymes to work. It can still cause gynecomastia in other people. In addition, just as progesterone increases s3xual desire in women, and often decreases with dramatic increase in its levels, high levels of progestogenic steroids can reduce s3xual desire in male bodybuilders. Yes, although there is a big difference in each individual. However, this activity is progestogenic and inhibits the production of LH, and contrary to popular belief, even a small amount of deca can greatly inhibit testosterone. To some extent, nandrolone produces a scent in estrogen, and it does not appear that this can be completely prevented by the use of aromatase inhibitors - in fact, aromatase may not be involved in this process at all (in humans). There is no evidence that this happens. Enzyme CYP 2C11 is, in my opinion, the most likely candidate for this project. In any case, Cytadren, an aromatase inhibitor, has not been found to be effective in inhibiting the aromatization of nandrolone. This medicine is effective at a dose of 400 mg per week. The half-life of nandrolone decanoate makes it unsuitable for short alternating cycles, but it is suitable for additional conventional cycles, which have a self-regulating effect in the weeks following the last injection. Common Name Nandrolone Formal Name Estr-4-en-3-one, 17-beta-hydroxy-CAS Registration No. 434-22-0 ATC Code A14AB01 Merck Reference No. 6391 Chemical Formula C18H26O2 Molecular Weight 274.40101 (% 10101)%) Metabolism Hepatic Half-Life Elimination 6 Days Abortion Pregnancy Stage X Intramuscular Management Methods
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