Clinicians used continuous electroencephalography (EEG) to analyze the seizure activity of a patient and prescribe the appropriate treatment. When the patient suffered a heart attack mid-recording and passed away, the scientists were able to record activity in a dying brain for the first time in history. The study of this patient, published in Frontiers in Aging Neuroscience, suggests that the brain might remain active during – and even after – death. In fact, it might be programmed to coordinate the entire process.
Multiple Sclerosis Is Likely Caused by a Virus, Finds Study of 10 Million Military Personnel
MS is a demyelinating neurodegenerative disease that, for many years, has been considered an autoimmune disease of unknown etiology. A large study using data collected over two decades of studying military personnel suggests that MS is a complication of infection by the Epstein-Barr virus (EBV), a herpesvirus that causes the childhood disease infectious mononucleosis. In the study of 10 million people, 800 of 801 MS cases in the sample group occurred in individuals who had previously tested positive for EBV. The research, by Harvard Medical School, was published in Science. The microbiome is the community of microbes, including bacteria, viruses and phage that live in a specific habitat. Microbiomes are found all over and inside our bodies. How the gut microbiome affects our metabolism of food and interacts with other body systems is of particular interest to researchers. A team working in mouse microbiome models explored how 10 different diets affect the secretion of a type of antibody, called IgA, by mucosal cells in the gut. The researchers found that a high-protein diet stimulated extra IgA secretion via a microbiome-mediated mechanism. The research was published in Nature Communications. The microbiome is the community of microbes, including bacteria, viruses and phage that live in a specific habitat. Microbiomes are found all over and inside our bodies. How the gut microbiome affects our metabolism of food and interacts with other body systems is of particular interest to researchers. A team working in mouse microbiome models explored how 10 different diets affect the secretion of a type of antibody, called IgA, by mucosal cells in the gut. The researchers found that a high-protein diet stimulated extra IgA secretion via a microbiome-mediated mechanism. The research was published in Nature Communications. The microbiome is the community of microbes, including bacteria, viruses and phage that live in a specific habitat. Microbiomes are found all over and inside our bodies. How the gut microbiome affects our metabolism of food and interacts with other body systems is of particular interest to researchers. A team working in mouse microbiome models explored how 10 different diets affect the secretion of a type of antibody, called IgA, by mucosal cells in the gut. The researchers found that a high-protein diet stimulated extra IgA secretion via a microbiome-mediated mechanism. The research was published in Nature Communications. The microbiome is the community of microbes, including bacteria, viruses and phage that live in a specific habitat. Microbiomes are found all over and inside our bodies. How the gut microbiome affects our metabolism of food and interacts with other body systems is of particular interest to researchers. A team working in mouse microbiome models explored how 10 different diets affect the secretion of a type of antibody, called IgA, by mucosal cells in the gut. The researchers found that a high-protein diet stimulated extra IgA secretion via a microbiome-mediated mechanism. The research was published in Nature Communications. Benzodiazepines are a class of drugs used to treat conditions such as anxiety and sleep disorders. However, for reasons not well understood, long-term use of benzodiazepines can cause physical dependence and even cognitive impairment. A study in mice demonstrated that sleep-inducing doses of diazepam administered over several weeks degraded the connections (synapses) between nerve cells in their brains, leading to cognitive impairment. These effects were eventually reversible, despite persisting for some time after diazepam treatment was stopped. The researchers believe these findings could have implications for the treatment of anxiety and sleep disorders in people at risk of dementia. The research was published in Nature
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